Neurotypical Disorder
Neurotypical Disorder, also called Neurotypical Personality Disorder and Herd Mentality, is a developmental disorder characterized by neurotypical behaviours such as:
*difficulty understanding people who have significantly different thought patterns or behaviours
*Inability to express oneself clearly, inappropriate use of language
*inapproriate use of laughing and noise
*restricted levels of understanding of deep topics
*poor listening skills
*repetitive consumption of false information
*strict conformance to social norms
*Needs to attend to social gatherings on discrete periods of time to reinforce the need of belonging to a group
*tendency for individuals to mimic the actions (rational or irrational) of a larger group
Not required but may also be present:
*belief in an imaginary higher power
*reliance on indirect communication
Neurotypical individuals often assume that their experience of the world is either the only one, or the only correct one. NTs find it difficult to be alone and are often intolerant of seemingly minor differences in others. When in groups NTs are socially and behaviorally rigid, and frequently insist on the performance of dysfunctional, destructive, and even impossible rituals as a way of maintaining group identity. NTs find it difficult to communicate directly, and have a much higher incidence of lying as compared to persons on the autistic spectrum.
Etiology
NTD is believed to be genetic in origin. Studies have shown a link between the GABRB3 gene and neurological atypicalism. This gene plays a role in GABA(a) receptors in the brain and is believed to be responsible for empathy. The heightened empathy is the mechanism through which the herd mentality is able to limit the person with this disorder from becoming self-actualized and "breaking from the herd".
Treatment
Medical Intervention
There currently is no medical intervention available to target the GABRB3 gene but CRISPR/Cas9 has shown efficacy with other genes as a gene therapy. There are gene knockout kits available for research but information on this is not currently available.
Behavioural Intervention
Additional studies have shown that “Copying what other individuals do can be useful in many situations, such as what kind of phone to buy, or for animals, which way to move or whether a situation is dangerous.
“However, the challenge is in evaluating personal beliefs when they contradict what others are doing. We showed that evolution will lead individuals to over use social information, and copy others more than they should.
“The result is that groups evolve to be unresponsive to changes in their environment and spend too much time copying one another, and not making their own decisions."
These results indicate that behavioural interventions might include:
*Teaching independence
*Teaching problem-solving skills
*Non-conformance training
Prevalence
No studies have been conducted to indicate presence of NTD, but studies that show prevalences of atypical populations, such as epileptics and others, may give a general indication by subtracting the atypical population from the total population to give an estimate.
*difficulty understanding people who have significantly different thought patterns or behaviours
*Inability to express oneself clearly, inappropriate use of language
*inapproriate use of laughing and noise
*restricted levels of understanding of deep topics
*poor listening skills
*repetitive consumption of false information
*strict conformance to social norms
*Needs to attend to social gatherings on discrete periods of time to reinforce the need of belonging to a group
*tendency for individuals to mimic the actions (rational or irrational) of a larger group
Not required but may also be present:
*belief in an imaginary higher power
*reliance on indirect communication
Neurotypical individuals often assume that their experience of the world is either the only one, or the only correct one. NTs find it difficult to be alone and are often intolerant of seemingly minor differences in others. When in groups NTs are socially and behaviorally rigid, and frequently insist on the performance of dysfunctional, destructive, and even impossible rituals as a way of maintaining group identity. NTs find it difficult to communicate directly, and have a much higher incidence of lying as compared to persons on the autistic spectrum.
Etiology
NTD is believed to be genetic in origin. Studies have shown a link between the GABRB3 gene and neurological atypicalism. This gene plays a role in GABA(a) receptors in the brain and is believed to be responsible for empathy. The heightened empathy is the mechanism through which the herd mentality is able to limit the person with this disorder from becoming self-actualized and "breaking from the herd".
Treatment
Medical Intervention
There currently is no medical intervention available to target the GABRB3 gene but CRISPR/Cas9 has shown efficacy with other genes as a gene therapy. There are gene knockout kits available for research but information on this is not currently available.
Behavioural Intervention
Additional studies have shown that “Copying what other individuals do can be useful in many situations, such as what kind of phone to buy, or for animals, which way to move or whether a situation is dangerous.
“However, the challenge is in evaluating personal beliefs when they contradict what others are doing. We showed that evolution will lead individuals to over use social information, and copy others more than they should.
“The result is that groups evolve to be unresponsive to changes in their environment and spend too much time copying one another, and not making their own decisions."
These results indicate that behavioural interventions might include:
*Teaching independence
*Teaching problem-solving skills
*Non-conformance training
Prevalence
No studies have been conducted to indicate presence of NTD, but studies that show prevalences of atypical populations, such as epileptics and others, may give a general indication by subtracting the atypical population from the total population to give an estimate.
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